Sentence examples for clones we tested from inspiring English sources

After 8 to 12 weeks, all clones we tested developed teratoma containing various tissues including gut-like epitheliums (endoderm), cartilages (mesoderm) and neural rosettes (ectoderm) (Fig. 2E).

Four out of five fosmid clones we tested harbour cross-species functionality for the gene assayed, and three out of the four rescue a RNAi phenotype in Drosophila melanogaster.

All the PαS-derived clones we tested had immature ES-cell-like characteristics, according to their gene expression, in vivo pluripotency, and the competency to produce adult chimeric mice.

Since a large portion of cDNA from OHCs was derived from mitochondrial genes [ 66] (55% of known gene clones), we tested whether these mitochondrial clones were false positives, showing His+ and lacZ+ phenotypes, independent of any interaction with prestin.

Interestingly, most of the RNAi clones we tested caused some degree of disruption of PAT-4/ILK expression (i.e., in M-lines and dense bodies in body-wall muscle cells) when fed to animals from hatching (Fig. S2, Table S3, Supporting information), suggesting that their thermotolerant phenotypes may be mediated, at least in part, via pat-4/ILK.

To confirm binding between Dsh and this putative partial-length Hipk clone, we tested whether an interaction could be recapitulated by in vitro pull-down assay.

A similar reduction in clump-size and restoration of mating was seen in six of the other seven evolved clones that we tested.

For clone E6-1, we tested the phenotype of introducing the wild type versions of the genes that harbored the eight putative causative mutations, as discussed above.

No significant difference in migration capability between the two clones was detected.Next, we tested the hypothesis that the reduction of CXCL12 by the COUP cells is important for their migration toward a CXCL12 gradient.

After cloning the fragments into pGEM T-easy vector, we tested clones for positive inserts using restriction analysis and sequenced the fragments in random clones.

Due to mapping discrepancies of one SNP and failure to clone one promoter, we tested only four out of the seven EMSA-positive SNPs identified thus far.