Sentence examples for clones we found from inspiring English sources

By utilizing two functionally distinct human MSC clones, we found that so-called "pericytic" MSCs secrete the pro-angiogenic vascular guidance molecule SLIT3, which guides vascular development by directing ROBO4-positive endothelial cells to form networks in engineered tissue.

By molecular bar coding and tracking individual clones, we found that, although AZA alters the sub-clonal contribution to different lineages, founder clones are not eliminated and continue to drive hematopoiesis even in complete responders.

By constructing chimeric Envs and fine mapping between sensitive and resistant Env clones, we found that substitution of highly conserved isoleucine (I) with methionine (M) (ATA to ATG) at position 424 in the C4 domain conferred enhanced neutralization sensitivity of Env-pseudotyped viruses to autologous and heterologous plasma antibodies.

In CSN5-mutant clones, we found 17 and 11 examples for each group, respectively.

Out of the ∼600 screened ES clones, we found three clones in which the Danish mutation was inserted in one of the BRI2 alleles.

Out of ∼600 screened ES clones, we found two targeted clones for T668A and four clones for Y682G mutation (Fig. 1B).

In this subset of clones, we find that full and partial restorations explain 44.7 and 14.8%% of observed phenotypic variation, respectively.

Using high-resolution mapping and positional cloning, we found that the fh131 mutation disrupts the Nance-Horan syndrome-like 1b (nhsl1b) gene.

In fact, in any selected clone, we found cells that expressed only one of the molecules (CD70 or CD80) and cells that expressed the two molecules.

By positional cloning we found a heterozygous mutation L95F in the Kelch-like homologue 9 gene, encoding a bric-a-brac Kelch protein.

Within the 21 mutant transcripts cloned, we found several different mutations, most of which show features of aberrant splicing leading to frameshifts and premature stop codons (Additional file 1: Figure S5).