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In this work, three classes of mechanisms that can cause load sequence effects on fatigue crack growth are discussed: mechanisms acting before, at or after the crack tip.
It is shown that there are five classes of mechanisms, including surface vertical cracks, opening interface cracks, sliding interface cracks, substrate deformation and macroscopic cleavage or spallation.
The methodology presented in this paper is restricted to single-degree of freedom function generation mechanisms and it can be extended to other classes of mechanisms.
These two classes of mechanisms are not the only ones that have been proposed for applying in the development of mathematical reasoning.
Two general classes of mechanisms, those defined by the user and those dictated by the endogenous cells and tissues, can control dynamic hydrogel microenvironments.
Detailed evaluation of the relative importance of these two classes of mechanisms shows that they compensate nearly exactly at moderate strains, so that volume strain is very small in the first stage of the tensile tests.
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We also grouped the observed events in the Philippines (Fig. 1b) and Japan (Fig. 8b) into the four classes of mechanism on the basis of the NIED estimated CMT mechanisms and calculated the normalized frequency distributions of the source amplitude ratios for each class (Fig. 13).
The high fraction of GWAS loci with proposed mechanisms suggests that these classes of mechanism play a major role.
Two main classes of mechanism have been proposed to account for the way in which self-motion information influences neuronal spatial representations: continuous attractor models and oscillatory interference models, which we review below.
Two classes of mechanism could explain our data: the iRhoms might either inhibit the function of active rhomboid proteases, for example by a dominant-negative effect, or could act directly on substrates, destabilizing them and preventing them from being cleaved by proteases.
The geometric model of this class of mechanisms is first introduced.
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