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The focus on solely CDH patients, with their compromised circulation, may explain this high incidence.
This absence of continuous circulation may explain why H1N1 was not the dominant subtype before the emergence of the pandemic H1N1/09.
The decrement in cardiac output and disturbance in systemic circulation may explain the limitations in somatic growth seen in these fetuses and impaired neurodevelopmental outcome [ 32– 32].
In addition, a great number of activated neutrophils during extracorporeal circulation may explain the decreasing sRAGE levels [ 27]. sRAGE is known to have multiple protease-sensitive sites, so it might be degraded by neutrophil-derived proteolytic enzymes [ 28].
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Taken together, these findings seem to indicate that PCP may impede the transfer of maternal T4 to the fetus across the placental barrier to a certain extent by inhibiting maternal T4 binding to TTR. is mechanism may also have favored the transport of maternal PCP to the fetal circulation, which may explain the higher concentration of this compound in cord blood compared with maternal blood.
IFNγ + T-cells are found in inflamed lesions in GPA and recruitment from the circulation may therefore explain the reduction during the active phase of the disease [ 10, 42- 44].
The closer anatomical proximity of the nasopharynx to a smaller brain and the existence of the rete mirabile in pigs relative to humans as well as the preserved cardiac circulation in stroke patients may explain these different findings.
Because pulmonary infiltrates are uncommon in previously healthy persons with ILI, a simultaneous circulation of other respiratory pathogens may explain our observation.
Lactate within the muscle cell is rapidly removed or consumed when circulation is restored [ 52], which may explain why lactate in the muscle sample taken 1 hour after standing had decreased or remained at anaesthetic levels.
The posterior circulation is more densely innervated by the trigeminal system, which may explain the frequency of headache in posterior circulation infarcts.
Different immunological activity in the central lymphoid system (reflected in the circulation) versus sites of inflammation in tissue may explain this.
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CEO of Professional Science Editing for Scientists @ prosciediting.com