Sentence examples for circulation compensatory from inspiring English sources

In response particularly in chronic hypoxia [ 14] foetal circulation compensatory mechanisms involve nitric oxide (NO) synthesis by endothelial nitric oxide synthase (eNOS), which eventually contributes to blood vessel tone regulation.

However, the markedly increased immunoreactive leptin level in circulation suggests a compensatory mechanism to increase the demand for leptin to regulate the energy imbalance.

There is a disparity between the number of Tregs found in the peripheral blood of RA patients compared to the numbers of Tregs isolated from the joint of the same individuals, with more Tregs in the inflamed joints than in circulation, suggesting that compensatory mechanisms take place at the site of inflammation [ 46, 47].

Surgical treatment can be complicated due to the compensatory collateral circulation through meningeal and leptomeningeal anastomosis.

Unlikely nondiabetic critical limb ischemia presentations, diabetic neuroischemic wound territories show major collateral deterioration, 20– 22, 32 with no opportunity for a compensatory collateral circulation.

In the initial days after acute blood loss, the mobilisation of erythrocytes from storage organs such as the spleen, liver, subcutaneous tissue, great vessels and pulmonary circulation occurs as an urgent compensatory measure [ 17, 18].

Thus, a compensatory mechanism for inadequate blood circulation is expansion of the blood volume.

Haematocrit and haemoglobin concentrations in the blood were unaffected by SWCNT exposure; and the absence of changes in the red and white pulp of the spleen excluded a compensatory haematopoietic response to protect the circulation.

It is currently unclear whether increased PEDF levels in the circulation play a pathogenic role, or are a compensatory response to angiogenic diseases.

The loss of agonist responses may represent a compensatory mechanism to protect the pulmonary circulation from changes in diabetes that promote pulmonary vasoconstriction.

Increase in kidney IGF-I levels due to its increased trapping from the circulation was hypothesized to be a key mediator of compensatory renal enlargement.