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In AATD COPD patients (i.e. AATD + AUG and AATD-AUG groups) we find no evidence to support a correlation between circulating arterial cytokines and the degree of airway obstruction.
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The present study was designed to evaluate the relation between circulating adiponectin and arterial stiffness parameters, such as pulse wave velocity (PWV) and aortic augmentation index (AI), in patients with hepatic steatosis.
This was illustrated by increased plasma I-FABP levels in the venous outflow of the reperfused jejunum, when compared to arterial circulating levels and I-FABP levels prior to the IR period (* p<0.001. Fig. 2A).
Second, the causal relationship between reduced circulating EPC and arterial stiffness could not be confirmed.
Circulating leukocytes and arterial blood pressure were quantified as previously reported (19).
Nevertheless, further studies are needed to clarify the clinical effects of the circulating adiponectin on arterial stiffness.
Four main mechanisms are involved in effective circulating volume and arterial blood pressure restoration in septic shock [ 32].
The purpose of this study was to investigate the relationships between glycemic control with circulating EPCs and arterial stiffness in patients with type 2 DM.
In patients with type 2 DM, the level of circulating EPCs and arterial stiffness were closely related to their glycemic control.
Abbreviations as in Table 1 Our results demonstrate a close inter-relationship between glycemic control, circulating EPCs and arterial stiffness in patients with type 2 DM.
Although Yang et al. [ 34] reported that impaired EPC activity was associated with reduced elasticity among hypertensive subjects, no significant correlation was again demonstrated between the number of circulating EPCs and arterial elasticity indices.
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