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Cholesterol crystal embolism (CCE) is caused by small crystals of cholesterol dispersed from atherosclerotic plaques of the aorta.
Similarly, quercetin also inhibited cholesterol crystal induced NLRP3 inflammasome activation.
Mechanisms and factors associated with the nanoparticle-mediated modulation of cholesterol crystal behaviour are proposed.
Authors hypothesize that specially designed nanoparticles may therapeutically modulate cholesterol crystal behaviour in atherosclerosis.
Proposed mechanism of spontaneous cholesterol crystal formation in the organism is discussed.
This article introduces a hypothesis on nanoparticle-mediated modulation of cholesterol crystal behaviour in the atherosclerotic plaques.
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For this purpose, we crystallized unesterified cholesterol from hot ethanol to obtain cholesterol crystals.
Underneath the fibrous tissue are often softer materials containing cellular components and cholesterol crystals.
Feasibility of the assessment of cholesterol crystals in human macrophages using micro optical coherence tomography.
Over time, bio-mineralization (calcified regions) of a plaque may occur and cholesterol crystals may penetrate into the fibrous tissue.
The role of cholesterol crystals in progression of atherosclerosis is emphasized.
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