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IGF has been shown to be associated with myopia in a chick model.
Recently, established cadmium (Cd -induced omphaloCd -inducedmomphaloceleen used to investigate the pathogenesis of omphalochick
We designed this study to investigate the messenger RNA (mRNA) levels of TGF-β2 and TGF-β3 in the Cd-induced omphalocele chick model during early embryogenesis.
An experimental study was designed to investigate the effects of the lipid or water soluble subfractions of meconium on the intestines of gastroschisis in a chick model.
The present study was designed to establish a chick model for the mechanisms of SM on neurobehavioral teratogenicity, free of confounds related to mammalian maternal effects.
We designed this study to investigate the hypothesis that Eya1 and Eya2 gene expression is down-regulated during the critical period of early embryogenesis in the Cd-induced omphalocele chick model.
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These distinct results may reflect the difference between mouse and chick models, or differences between the in vitro and in vivo approaches.
Currently, chick models play a pivotal role in animal research as an alternative and outbreed experimental species to humans.
We found that CGP46381, a GABAB receptor antagonist, which inhibits experimental myopia development in chick models [ 12], also inhibits FDM in guinea pigs.
There is considereable crosstalk between the BMP and FGF signaling pathways in chick models; FGF signaling maintains a pool of undifferentiated stem cells.
Our data suggest that FGF2-induced BMP2 and BMP4 could work either in the autocrine or paracrine mechanisms in cooperation with FGF to elicit cell-cycle exit as shown in chick models of lens cell-cycle exit-couple differentiation (Boswell et al. 2008a, b; Jarrin et al. 2012).
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