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That this medically most relevant group of genes is the one least characterized appears problematic.
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The degree of condensation of VOx species on the mixed oxide surface, including some possible epitaxial grafting, even if not clearly characterized, appeared to play a determining role in oxidative dehydrogenation properties.
Given that all WSSV isolates genetically characterized appear to have a very recent common ancestor, the consensus view is that during WSSV spread throughout Asia the DNA genome has been progressively shrinking [3], [8], [9], [10], [11], [12], [16], [17].
The pathogenesis of MM as of all changes in migraine related to hormones has not been fully characterized but appears to be related to estrogen withdrawal [19, 64].
Time pattern of this phenomenon has been characterized: dysfunction appears usually on day 1 roughly in two-thirds of affected patients, on day 2 3 in the other third, while recovery takes 7 10 days.
Although the full complement of populations is not completely characterized, it appears they can be represented by SSTF "expression codes".
Although the pathophysiology of diabetic endothelial dysfunction is incompletely characterized, it appears to be multifactorial.
Although the killing factor produced by skfA-H operon has not been completely characterized, it appears that its activity is similar to that of bacteriocins.
Additionally, an N-acylethanolamine-specific phospholipase D (NAPEPLD, Q6IQ20) has been characterized, which appears to have a role in the generation of endocannabinoids/endovanilloids, including anandamide 246.
In plant species where retrotransposons have been characterized, it appears to be lineage specific whether Ty1/ copia-like or Ty3/ gypsy-like retroelements are more abundant in the genome, likely because retrotransposons can proliferate or be lost from genomes over relatively short evolutionary time scales [ 85, 87].
The Ds/Fat/Fj module has been most extensively characterized and appears to signal via formation of heterodimers with the extracellular domains of Ds and Fat (which are atypical cadherins), putatively in a gradient across the tissue (Ambegaonkar et al., 2012; Bosveld et al., 2012; Brittle et al., 2012).
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