Sentence examples for channels in brain from inspiring English sources
The emergence of flexible information channels in brain networks is a fundamental question in neuroscience.
Schwartz-Bloom, RD, Cook, TA, and Yu, X. "Inhibition of GABA-gated chloride channels in brain by the arachidonic acid metabolite, thromboxane A2". Neuropharmacology 35.9-10 (1996): 1347-1353.
By means of patch clamp techniques, it was shown that actin microfilaments, which formed the structure of the cortical cytoskeleton, participated in the regulation of chloride ion channels [8, 9], Na+/K+-ATPase [10], voltage-gated sodium channels in brain cells [11], and sodium channels in the cells of polar reabsorption epithelium [12].
MCP-1 also affects voltage-gated potassium channels in brain endothelial cells [ 70].
The membrane pore protein AQP4 comprising the major water channels in brain is clustered together with the DGC via interaction between AQP4 and α-syntrophin [ 29], which in turn binds to dystrophin and β-DG.
Only in the last few years has it become clear that the antibodies are not mainly directed against the voltage-gated potassium channels themselves in these patients, but against other proteins that are complexed with the voltage-gated potassium channels in brain tissue (Irani et al., 2010; Lai et al., 2010).
Schwartz, RD, Wess, MJ, Labarca, R, Skolnick, P, and Paul, SM. "Acute stress enhances the activity of the GABA receptor-gated chloride ion channel in brain". Brain Res 411.1 May 12 , 1987): 151-155.
AQP4, the primary water channel in brain, is expressed on ependymal cells and astrocytes.
Electrophysiological studies showed that U50488 could inhibit the P-type calcium channel in brain Purkinje cells through activation of κ-opioid receptors [ 15, 16]; it could also inhibit the L-type calcium, sodium, and potassium channels in ventricular myocytes directly at a higher concentration [ 17, 18].
Figure 3 Recovery results from eight-channel in-vivo brain data with the subsampling ratio (pmb{R=2,4}).
Image caption Researchers say the protein "provides exceptional levels of protection for eight hours after stroke onset" They found that the protein blocked acid-sensing ion channels in the brain - something the researchers say are key drivers of brain damage after stroke.
