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Significant changes of sodium channel expression during development were detected.
Indeed, blood pressure per se appears to promote CaL channel expression in small arteries, and even short-term rises in pressure may alter channel expression.
Kir4.1 channel expression is found in the brain, inner ear, eye, and kidney.
We also examined chloride channel expression before and after wounding.
Human brain served as a positive control for channel expression.
In neurons of the same type, is ion channel expression highly variable and/or is there any relationship between ion channel expression that is conserved?
This dysregulation may be the result of changes in channel expression levels, post-translational modulation and/or variant channel expression and dysfunctional channel assembly.
Thus, Kv channel expression seems to be not only region, but also species specific.
Can we relate channel expression to the two current components that we have described?
To assess levels of endogenous channel expression, oocytes were injected with dH20 only.
Channel expression into Xenopus oocytes and two-electrode voltage-clamp recordings were performed as described [27].
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