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The ability to synchronize ones physiology to anticipate environmental changes is thought to be the driving force behind the evolution of a network of circadian oscillators that adapt and respond, and yet have the ability to "keep time" in absence of any external cues.
This multiplicity of changes is essentially like that of the evolution of de novo genes, and expands the point from the de novo genes section, because now the multiplicity of changes is thought to happen in each of many genes that are part of a network.
The capability to reset methylation changes is thought to provide flexibility of the body to regulate the timing of gene expression during critical periods of growth and development, and in this light it is not surprising that imprinted genes tend to be found in evolutionarily conserved imprinted domains [ 21].
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These changes are thought to be primarily a result of decreased testosterone levels, although changing social roles for women in midlife may also be a factor.
Many of these genomic changes are thought to represent driver mutations in oncogenesis.
The larger morphological changes are thought to contribute to the concentration dependence of diffusion making free volume theory inapplicable.
These changes are thought to be the reason for the enhanced on-rate of the mutated DARPin.
These changes are thought to exacerbate project content factors and to raise the value of NPD leadership and integration techniques.
These changes are thought to be due to the deprotonation of the 4-amino moiety of the naphthalimide fluorophore.
These changes were thought to be associated with extinction to cocaine-associated environmental cues.
These mucosal changes are thought to be due to luminal stasis.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com