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On the other hand, in 2011 Yang et al. analyzed the transcriptional response of Pseudomonas stutzeri exposed to chemically-synthesized QDs and observed changes in the transcription profile of 7 genes including some denitrification genes (narG, napB, nirH and norB) and the up-regulation of the superoxide dismutase gene (sodB), suggesting the production of ROS [ 25].
In vivo studies indeed suggest that IL-6 is an inducer of gliosis in the retina through changes in the transcription profile of glial cells [8].
The study was designed to reveal the possible pathways by examining changes in the transcription profile after H2O2 treatment with both the cap1-deleted strain CJD21 and its parental strain CAI4 using microarray analysis.
Microarray analysis revealed drastic changes in the transcription profile of E. coli L-forms, which included the overexpression of many genes involved in stress responses and of unknown function that may be involved in the formation and survival of L-forms.
We also investigated the changes in the transcription profile of embryos which were smoke-treated for 3 and 6 h, after a 3 h delay.
The purpose of this study was to generate a global mRNA expression profile of myo1Δ strains using yeast oligonucleotide microarrays to identify potential relationships between the multiple myo1Δ phenotypes mentioned and changes in the transcription profile of specific genes that can be functionally related to these phenotypes.
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We observed a significant change in the transcription profile of hepatic cells when APAP was co-administered with IFN-β.
This change in the transcription profile was also reflected by the elevated total enzyme activities for l-arabinose reductase, l-arabitol dehydrogenase, and xylitol dehydrogenase in the cell free extracts.
These clusters indicate the occurrence of major changes in the transcription profiles between the bent-cotyledon to the fully-developed embryo stages of seed development (25 35 DAP).
This change would be initiated by a shift in the transcription profile, followed by a delayed change in protein expression profiles.
To determine how cardiac mIGF-1 expression changes the transcription profile of the pre- and post- infarct epicardium, we compared the expression of mIGF-1 transgenic to wildtype epicardial samples in normal and infarcted hearts.
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