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The junctions are not open pores but dynamic channels, which change their permeability with changes in cellular activity.
No significant changes in cellular viability, as judged by production of formazan were observed (Table 1).
Severe osmotic stresses, salinity, drought, and cold, cause detrimental changes in cellular components.
Cells were collected and extracted with lysis buffer to detect changes in cellular protein levels.
The variability in temperature profiles maybe related to changes in cellular control factors.
These drastic changes in cellular architecture ensure the proper segregation of chromosomes and inheritance of organelles.
Chronic exposure to ethanol may results in pathophysiologic changes in cellular function.
Secondary analyses of urine samples assessed pre-post changes in cellular biochemistry, neurochemistry, and microbial metabolism.
FTIR analysis indicated that the ITCs caused changes in cellular components comparable to vinblastine.
At M-phase exit, these major changes in cellular architecture must be reversed.
Changes in cellular NAD+ levels can occur due to modulation of pathways involved in NAD+ biosynthesis and consumption.
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