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The altered cytokine levels and subsequent changes in airway redox status lead to goblet cell metaplasia, transforming airway epithelial cells into mucus secreting cells, which release overwhelming amounts of mucin, especially mucin5AC, which obstructs the smaller alveoli and causes obstruction in breathing in asthma [5] [10].
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This might be due to changes in airway mechanics in the presence of inflammation or structural changes within the airways.
The purpose of this study was to evaluate changes in airway infantile hemangioma treatment.
This may involve changes in airway smooth muscle function.
Changes in airway measurements were assessed using lateral cephalometric radiographs.
The changes in airway measurements are shown in Tables 2 and 3.
These changes in airway structure result from chronic inflammation and airway remodeling (Woolcock and Permutt 2011).
In chronic airway inflammatory lung diseases, airway remodeling is correlated with marked changes in airway mechanics and symptoms related to airway obstruction [ 14].
Minimal changes in airway eosinophils are predicted in this phase.
Airway diameter with changes in lung volume, or so-called airway distensibility is reflected by reactance, and changes in airway pressure with lung volume by resistance.
Changes in airway pressure and TV were not quantitatively related to cardiac index changes.
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