In addition, BHE conferred protection against liver damage as evidenced by the marked decrease in lymphocyte infiltration, hydropic degeneration, fatty changes, central vein congestion, Kupffer cell hyperplasia, and necrotic changes.
The possible influence of such cellular changes on vein graft failure is not well understood.
Notably, treatment with BHE at doses of 500 and 1000 mg/kg reduced the fatty changes, central vein congestion, and necrosis more than in the silymarin-treated group.
Similarly, recurrent DVT was diagnosed by measuring changes in vein diameter over time, defined as an increase in thrombus diameter to 4 mm in a previously affected vein segment, as opposed a reduction or no change in size [ 25].
CCl4 induced severe loss of the hepatic architecture, marked hydropic degeneration, fatty changes, central vein congestion, Kupffer cell hyperplasia, necrosis, and scattered lymphocytes in between the hepatocytes and in the sinusoids.
In order to avoid changes in vein diameter unrelated to respiratory variation, gentle pressure by the US probe was used to collapse the IJV in order to distinguish it from the carotid artery, then the pressure was relieved to the US probe-skin interface and attention was given to avoid influence of probe compression on IJV dimensions [ 13] during the US examination.
In obese patients, the respiratory change of the vein diameter was smaller, and the vein was deeper.
Objectives: To study the histological changes of veins and venous malformations after injection of pingyangmycin and sodium morrhuate.
The visibility of venodilation correlated with the extent of change of the vein size during a respiratory cycle but not with the end-expiratory or end-inspiratory vein diameter.
Change in basilic vein size was modeled using mixed model analysis with a Tukey correction for multiple comparisons to determine if significant differences existed between different maneuvers.
