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After 24 h of Aβ treatment in the Cx chamber we found that phosphorylation levels of Tau Thr231 increased in post-synaptic hippocampal neurons.
Using a parallel plate flow chamber, we found that depending on P-selectin incubation concentration untreated cells incurred up to a 6-fold increase in rolling velocity while subjected to an approximately 10-fold increase in biologically relevant shear stress.
When we added plasma samples from ApoE−/−FNfl/fl and ApoE−/−FNMxCre mice to the lower part of the migration chamber, we found that plasma from ApoE−/−FNfl/fl mice induced twofold higher migration rates of vSMCs than plasma from ApoE−/−FNMxCre mice (Fig 7D).
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When plotting the amount of product inside the chamber relative to the total amount of peptides in the chamber, we find that Suc-LLVY-MCA and Bz-VGR-MCA are cleaved immediately once inside the chamber (90% of peptides are products).
Using antibodies against mitotic markers Phospho-Histone 3 (PH3) and Cyclin B in sav mosaic egg chambers, we found that the PFC clones showed prolonged oscillating patterns of PH3 and Cyclin B expression after stage 6 (Fig. 3B,D).
By using both an optimized permanent magnets and the axial electromagnet around the chamber wall, we found the improvement of polysilicon etch uniformity in addition to the increase of polysilicon etch rate due to the combination of the better features of both magnets.
In growth chamber studies, we found support for this hypothesis: only grass-associated CYDV-RPS stunted the shoots and crowns of Panicum virgatum L. (switchgrass), a perennial native North American prairie grass, whereas crop-associated BYDV-PAV (and coinfection with BYDV-PAV and CYDV-RPS) most stunted annual Avena sativa L. (oats).
Finally, using a Boyden chamber assay, we found that Wnt3a promotes cell migration (Supplementary Figure S3C).
By using Scarification test and Transwell chamber assay, we found that WM130 could attenuate the migratory and invasive capacities of HSC-T6 cells.
Using a modified Boyden chamber assay, we found that blockade of autocrine TGFβ signaling significantly inhibited motility of both DNRII and DNRII-EGFP cells in comparison with their respective controls.
We, therefore, interpret the observed dry-bone fracture patterns to be due to post-depositional sediment movement within the chamber as Units 2 and 3 are reworked, as well as unintentional damage by cavers or others entering the chamber; and we find no evidence for green fractures associated with trauma in the H. naledi fossil assemblage.
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