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However, some variables were unavoidably excluded from the analysis of CES and non-CES groups because of the characteristics of each group; that is, AF, LV systolic dysfunction, and the presence of a CES were excluded from the analysis of the non-CES group, and the presence of a CES was excluded from the analysis of the CES group.
As shown in Table 1, the non-CES group had a significantly higher frequency of hyperlipidemia, smoking, and heavy alcohol use, while the CES group had a significantly higher prevalence of previous antithrombotic therapy and ischemic heart disease.
Additionally the CES group showed significantly less muscle atrophy in both muscles compared to the COM group (P < 0.05 0.01).
However, motor function remained significantly worse than preoperative values in the CES group (−69.08 ± 5.94; n = 6; P < 0.01).
With consideration of the muscular atrophy and the percentage loss of functional axons, target organ reinnervation is still superior in the ANT group, although in the CES group no significant impairment of motor function recovery was detected.
As the pinch-test response to the 5th toe did not differ significantly between them, it can be concluded that pain perception recovery was retarded in the CES group but not less complete than in the ANT group.
Similar(51)
Extensive genetic approaches in C. elegans have highlighted that engulfment genes, collectively belonging to the ced group (cell death abnormal) [2], act along two distinct and parallel pathways converging towards the same end-effectors.
Mann-Whitney U test were computed to compare the performance of vermis CE group (n = 5) vs. C and hemispheric CE group (n = 8) vs. C.
The remaining tissue samples were included in the control CE group.
In the stratified analysis, excess risk was absent in the higher CE group (≥1.0 ppm).
In CE group the dentate-affected children were: 4 with hemisphere and 2 with vermis location.
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