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Alternative mechanisms how introns can delay expression of a certain transcript are intron retention and post-transcriptional splicing, which regulate RNA abundance as well as protein translation in certain cellular contexts.
Once samples are extremely diluted, stochastic events may lead to variations in the number of molecules of a certain transcript in aliquots of the same RNA preparation.
Certain transcript isoforms characterizing these processes are potential targets for breeding for drought tolerance.
The 14-3-3s 14-3-3s 14-3-3se also subject to the regulation by certain transcript familys [ 9].
This method permits the rapid testing of a large number of different tissues for the presence of a certain transcript.
Additionally, alternative splicing employed by numerous genes may enable certain transcript variants to escape from miRNA-mediated regulation.
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In particular, how certain transcripts are activated/maintained despite suppressed transcription during low body temperatures is poorly understood.
Myc mutants lacking transcriptional activity could up-regulate mRNA cap methylation of certain transcripts, implying that Myc proteins have a relatively direct effect on mRNA cap methylation.
Certain transcripts could escape cleavages due to the absence of intronic miRNA binding sites within these sequences.
The recruitment of enolase by the RNA degradosome has been implicated in the turnover of certain transcripts, and it is mediated by a small segment of roughly a dozen residues that lie within a natively unstructured sub-domain of RNase E. Here, we present the crystal structure of enolase in complex with its recognition site from RNase E at 1.6 Å resolution.
Certain transcripts correspond to multiple probe sets on the Affymetrix array.
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