Sentence examples for cerebral inflammation from inspiring English sources

Cerebral inflammation, apoptosis and neuronal survival was determined at day 10.

A20 loss elevates the levels of NF-κB-regulated inflammatory cytokines and causes spontaneous cerebral inflammation [17].

These results are exciting in that these agents might be useful candidates in treatment of cerebral inflammation.

Aim: [18F]-FEPPA, a radiotracer specific for TSPO (18 kDa translocator protein), is used in PET imaging (Positron Emission Tomography) as a biomarker of cerebral inflammation.

Nevertheless, over the last decade, TSPO has become an attractive target for positron-emission tomography (PET) imaging of cerebral inflammation due to its high expression after pathologic situations despite the low expression showed in the healthy cerebral parenchyma [5].

He explains that he is dying of a cerebral inflammation that developed after his surgery.

However, activated glial cells acquire neurotoxic properties, notably by releasing nitric oxide (NO) and glutamate [ 10, 11], in circumstances that include cerebral trauma, inflammation and infection.

Our results suggest that the reduction in ferroportin levels are likely associated with cerebral ischaemia, inflammation, the loss of neurons due to the well-characterised protein misfolding, senile plaque formation and possibly the ageing process itself.

Hypoxia can be a consequence in some pathological conditions, such as cardiovascular and cerebral ischemia, inflammation, pulmonary disorders and cancer, as well as in many physiological conditions including high altitude and physical exercise [ 2].

In one patient with increased CBFv, corresponding arterial narrowing on DSA, and cerebral ischemia, inflammation was seen surrounding middle and small sized blood vessels on autopsy as a possible underlying reason for the vascular alterations found in vivo.

In conclusion, as vascular damage with associated ischaemic change is widespread in AD brains it is likely that the reasons for a reduction in ferroportin levels are multifactorial but include cerebral ischaemia, inflammation, the loss of neurons due to the well characterised protein misfolding, senile plaque formation and possibly the ageing process itself.