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To restore difficulty, despite the central presentation, the stimulus was presented briefly (70 or 80 ms), and was followed by a post-mask.
This ipsilateral response was also faster than that from central presentation.
We cannot exclude an amygdala activity for central presentation, as reported in previous neuroimaging studies.
Table 1 reports these structures across the three time windows, for central presentation.
For central presentation, emotion separation was seen in the early latencies of 57 64 ms, in the right amygdala.
The latency was significantly shorter for upper visual field presentation than for central presentation (p<0.00001) and lower visual field presentation (p<0.003).
Similar(39)
Further, a strategy based on such effect could also explain the higher improvement in all groups for auditory stimuli presented in peripheral location compared to central presentations.
The current study replicated Shimojo et al.'s (2003) design, whilst controlling for potential inter-stimuli interference in central presentations.
The different brain regions sensitive to facial expressions in the first 130 ms are right-sided when the presentation is peripheral but are left-sided for central presentations.
For central presentations, four ROIs were defined for bilateral dorsal and ventral V1/V2 while for quadrant presentation, one ROI was defined for the activated part of the calcarine sulcus, e.g., for UL presentation, and only one V1/V2 ROI was defined for the right ventral area.
In the current study, synchronous central audiovisual presentation enhanced activity in the left pSTS region compared with unisensory presentation and this was more pronounced for stimuli with predictable than unpredictable timing.
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