This apparent paradox may be explained by the cellular expression of CXCL12 not providing a true reflection of its bioavailability, which depends principally on the presence in the tumor microenvironment of factors capable of disrupting CXCL12 from glycosaminoglycans [ 46].
First, the cellular expression of CXCL12 may not provide a true reflection of its bioavailability, which depends principally on the presence of factors capable of disrupting CXCL12 from glycosaminoglycans in the tumor microenvironment [22].
They provide a reflection of cellular redox activity which is principally a product of cellular energy metabolism, which also provides the primary source of reactive oxygen species which are thought to be important in the pathogenesis of diabetic complications [ 22].
The present paper reports the results of an experimental study of the Mach reflection of cellular detonations.
For cellular foams, transmission and reflection tend to be negligible in the infrared region, and behave like black bodies.
An experimental study of the Mach reflection of cellular detonations over the wedge is reported in this paper.
Recently, reports raised concerns that neural differentiation of MSCs might be a reflection of cellular stress rather than cells entering a true differentiation programme [13], [14], [15].
The extent of reduction is a reflection of cellular activity, and can be measured by either colorimetric or fluorometric methods.
Glycome complexity is a reflection of cellular complexity and the collective tools of genomics, proteomics, lipidomics and metabolomics are required for functional characterization.
Thus, the aberrant HERV-W expression associated with complex human diseases such as MS [ 14, 15] and schizophrenia [ 16, 17] may be a reflection of cellular events acting on promoters of genes harboring HERV-W elements or on proviral LTRs.
