Chelation of cellular zinc ions after rapid stretch injury, however, increases cellular reactive oxygen species.
Silencing the hTERT gene reversed ImmSig expression, increased cellular reactive oxygen species levels, altered mitochondrial membrane potential and induced apoptotic and proliferation changes in immortalized cells.
The method developed a new platform for a reliable collection of information on cellular reactive oxygen species release.
Furthermore, we evaluated whether carvedilol inhibits PDGF-induced cellular reactive oxygen species (ROS) and the activation of mitogen-activated protein kinase (MAPK).
As a downstream effect of TrxR1 inactivation, ethaselen causes a dose-dependent thioredoxin oxidation and enhances the levels of cellular reactive oxygen species in A549 cells.
NADPH oxidase (Nox) family enzymes are one of the main sources of cellular reactive oxygen species (ROS), which have been implicated in several physiological and pathophysiological processes.
Surfactant gelation is decreased when ROFA-AEC are pretreated with inhibitors of cellular reactive oxygen species (ROS), or with a mitochondrial anion channel inhibitor, as well as when A549-rho0 cells that lack mitochondrial DNA and functional electron transport are investigated.
Production of reactive oxygen species was assessed with DCFDA, Cellular Reactive Oxygen Species Detection Assay Kit.
ROS production was evaluated using DCFDA, Cellular Reactive Oxygen Species Detection Assay Kit (Abcam, Cambridge, UK).
In this study, we evaluate the role of HMF on the regulation of cellular reactive oxygen species (ROS) in human neuroblastoma SH-SY5Y cells.
The authors utilized antioxidant, hydroxylated fullerene (fullerol), and FoxO1 (transcription factor) to reduce cellular reactive oxygen species (ROS) activities, and induce osteoblast differentiation.
