Sentence examples for cellular levels we from inspiring English sources

For visualizing AMPAR subtypes at the cellular levels, we generated gene-specific probes as follows: GluR1 antisense probe covering nucleotides 434 1090 of GluR1 mRNA (Genbank, NM_001113325.1); nucleotides 631 1394 (NM_001039195.1) for GluR2; nucleotides 766 1524 (NM_016886.3) for GluR3 and nucleotides 295 884 (NM_019691.4) for GluR4.

To investigate the role of autophagy in the human malaria parasite at the molecular and cellular levels, we first explored the role of ATG8 during the erythrocytic cycle.

As mutant p53 suppresses miR-27a expression and miR-27a, in turn, downregulates EGFR cellular levels, we sought to investigate whether mutant p53 regulates function of EGFR through miR-27a.

To further investigate changes in Na+ and K+ cellular levels, we utilized amiloride at 1 mM and lidocaine at 1 μM to alter ion fluxes in C. raciborskii T3 over 2 h of exposure.

To study the mechanisms leading to the nuclear phenotype of isoform P1-deficient myofibers on molecular and cellular levels, we generated a P1 isoform-deficient immortalized myoblast cell line, using a previously established protocol that is based on the isolation of myoblast cultures from mutant mouse lines crossed into a p53−/− background (20).

Since ITF2357 showed anti-fibrotic effects at the cellular level, we wanted to examine its functionality in an in vivo rabbit corneal fibrosis model established previously6.

To reconcile the gap between the reported models and molecular pathways at the cellular level, we have related the single-cell-level in-vitro nanotherapeutics incorporation and its apoptotic action as well as drug efficacy in in-vivo tumor model.

To further verify the chromosomal translocation events at the cellular level, we adapted the chromosome painting technology to mark chromosome 5 with red fluorescence and chromosome 7 with green22.

To determine the anti-fibrotic effects of ITF2357 at the cellular level, we used the classical in vitro model of corneal fibrosis where pHCSFs were stimulated with 1 ng/ml TGFβ.

In order to study the effects of forces at the cellular level, we utilized two hierarchical, in vitro systems to mimic traumatic injury to rat cortical neurons: a high velocity stretcher and a magnetic tweezer system.

So from the cellular level we learn why, for example, cancer risks are based not so much on genetic disposition but on where we live and what we eat, drink and inhale; why some rare cancers are, in fact, contagious; and why aging is "a correlate, not a cause, of most cancers".