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The low-density cellular layer was separated, counted and resuspended in 200 μl of physiological solution for immediate surgical implantation.
The clear plasma above the separated cellular layer was carefully removed without disturbance to the cellular layer.
Identification of the AOB external cellular layer was performed as described in Ben-Shaul et al. (2010).
The AOB external cellular layer was targeted by making a small craniotomy immediately rostral to the rhinal sinus and advancing electrodes into the AOB at a 30° angle.
Below this layer, a wide, more densely populated cellular layer was formed by neuronal bodies of different sizes, some of which had a pyramidal appearance.
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However, when the cellular layer is fully crushed, the effect of density variation on energy absorption vanishes.
Ultimately, the ameloblasts atrophy and the cellular layer is lost entirely from the mature erupted tissue (7).
However, a reeler-like phenotype characterized by profound dyslamination of all cellular layers was observed in double Pafah1b1+/− Apoer2−/− mice (Fig. 6).
The localization of the mentioned areas and cellular layers was shown in Fig. 1.
Appropriate cellular layering was observed in wild-type sections stained for Gravin (green) and nuclei (white).
The most obvious design flaw of the retina is that the cellular layers are backwards.
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