Sentence examples for cellular absorption from inspiring English sources

Exact(13)

A sample (200 μL) from either cellular absorption or transport experiments was mixed with 20 μL methanol containing the internal standard (ginsenoside Re, 0.498 μg/ml).

To further support our previous in vivo results in rats, the present in vitro study was designed to evaluate the effects of 2, 3, 5, 4′-tetrahydroxystilbene-2-O-β-D-glucopyranoside (TSG) on the cellular absorption and human liver microsome metabolism of emodin.

Two biochemical mechanisms were shown to interplay in the cellular absorption of Fe2+, the transported form of iron: a fast diffusional process after the transfer of cells into the medium and a slow component mediated by an Fe transporter and stimulated by the action of Ca2+.

In addition, he holds many food patents, including a super tasty nutritional drink that fuels your body and mental focus to its full potential named Jimmy Juice, with electrostatic balance that captures increased efficiency and accelerates cellular absorption.

These effects of high dietary Mg2+ are the consequence of the requirement for these other TRPM channels for intestinal and cellular absorption of Mg2+.

Endocytosis is a cellular absorption process of large molecules that is primarily used for the nonselective internalisation of fluid and protein into the cell.

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Similar(47)

Moreover, soon after nasal delivery of a drug it first reaches to the respiratory epithelium, where it absorbed into the systemic circulation by trans-cellular and para cellular passive absorption, or by transcytosis or endocytosis [ 47, 48].

Stimuli such as hypotonic low Cl− conditions that activate the WNK SPAK/OSR1 pathway will induce net cellular Cl− absorption by promoting phosphorylation, thereby activating the N[K]CCs and concurrently inhibiting the KCCs.

In contrast, stimuli that inactivate the WNK SPAK/OSR1 pathway, such as hypotonic high K+ conditions, will suppress cellular Cl− absorption by promoting dephosphorylation, thereby inhibiting the N[K]CCs and concurrently stimulating the KCCs.

Several suspected means of in vitro resistance to these mustard agents include cytokine production defects in the bone marrow milieu, altered drug delivery by transporters that effectively decrease cellular drug absorption, and an increase in effective DNA repair of mustard-specific lesions [ 3, 4].

A model was derived describing cellular convection during absorption accompanied by a first-order chemical reaction.

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