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Surprisingly, AQP1 and AQP2 proteins identified in urine exosomes via secretion through renal ductal cells were implicated in pathophysiological processes in urinary epithelial cells [13].
Finally, neuroglial cells were implicated in various pathological processes.
Th17 cells were implicated in the erosive stage of chronic arthritis independent of TNF-α [ 28].
Both antigen-specific antibody and CD4+ T cells were implicated in protection via adaptive immunity.
However, based on surface receptors on γδ T cells, functional changes of these cells were implicated.
Moreover, CD5+ Fas ligand positive Breg cells were implicated in immune regulation in collagen-induced arthritis by promoting apoptosis in arthritis-associated T cells 46.
Similar(51)
Promoting NF-κB-mediated gene transcription in CD4 + T cells is implicated in its immunomodulatory activity.
The cerebellum and specifically cerebellar Purkinje cells are implicated in tottering mouse attacks.
Surgery-induced angiogenesis of dormant avascular micrometastases and surgery-induced activity of single malignant cells are implicated.
Increased iNOS expression in glial cells is implicated in the etiology of CNS diseases by causing inflammation and cytotoxicity [20, 21].
Mast cells are implicated in EIB, but the characteristics, regulation, and function of mast cells in patients with EIB are poorly understood.
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