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The integrin specificity of this interaction between type I collagen and tumour cells was not identified, however.
In the case of PTPRK, expression in normal B cells was not identified by qRT-PCR and therefore the RNA-seq data could not be validated.
Canonical mechanisms, such as the regulation of E2F transcription factor family members by the retinoblastoma protein RB1, were included in the network even if literature support explicitly demonstrating the presence of the mechanism in lung-related cells was not identified.
There is no further follow-up on this finding reported and the HCMV antigen possibly causing the crossreactive antibodies (the antigen should be a virion component since HCMV does not replicate in mouse cells) was not identified and it is also not clear with which host protein the antibodies may crossreact and whether the antibodies have indeed the potential to induce autoimmunity.
Similar(56)
Although infiltrating tumor cells were not identified microscopically, we estimated 25% tumor contamination.
However, Nkx2 5-derived blood cells were Nkx2 5-derived in the peripheral circulation in adult Nkx2–5IRES-Cre/+; R26YFPR/+ mice.
However, Epstein-Barr virus-encoded small RNA-positive cells were not identified by in situ hybridization.
Although long-term transplantation resulted in significant functional and pathological improvements in APP/PS1 mice, stem cells were not identified by immunohistochemistry or PCR at the study endpoint.
Proteins corresponding to spots no. 12 and 13, detected only in control cells, were not identified.
B cells were not identified in the devil facial tumor tissue, however, APC were present.
Reed-Sternberg and/or Hodgkin cells were not identified, although focally, eosinophils were prominent.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com