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We compare conventional full-area BSF and PERC solar cells to future cell designs with a gallium phosphide heteroemitter.
This protective effect may be attributed to permanent structural and functional changes induced in the mammary parenchyma by the reproductive process, including exposure to pregnancy hormones, resulting in a lower susceptibility of epithelial cells to future carcinogenic stimuli [ 37, 38].
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Thus, Notch is activated in cells distal to the Dl-expressing cells (corresponding to future ball-producing cells), whereas the proximal cells (corresponding to socket-producing cells) have lower Notch-signaling levels.
The localization of Wnt-expressing cells to the future posterior pole is the earliest morphogenetic asymmetry detected in Amphimedon (Fig. 2C, D), occurring prior to the initial cell sorting event that creates the embryonic cell layers.
Repopulating lymphocytes, however, do not bring about resolution but replenish resolving tissues with the necessary cellular players (CD3, B1 cells, natural killer [NK] and γ/δ T cells as well as CD4+/CD25+ T cells) to control future innate immune-mediated responses.
In conclusion, using fission yeast as a model system we confirm that moderate levels of stress due to oxidative metabolism during the logarithmic growth may prepare cells to encounter future periods of starvation or inactivity, and that a MAP kinase pathway has an essential role in linking endogenous stress and the activation of a genetic fitness program.
This suggests that the interactions between the CaV1.2 channels act as a type of 'molecular memory' that may alter how the cells respond to future activity.
Considering that cells other than neurons are clearly involved in the pathogenesis of MS, we plan to add oligodendrocyte cell lines to future studies of mutant hnRNP A1.
Because the significance of insulin resistance and secretion could differ by ethnic group, parental history of diabetes, and obesity, we examined the heterogeneity of the relation of insulin resistance and β-cell function to future development of type 2 diabetes.
We tried to establish an indicator for the S1 response in leukaemic cells to guide the future clinical development of S1.
Ischemic preconditioning induces complex physiological adaptations to improve the tolerance of cells and tissues to future ischemic episodes.
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