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The full repertoire of regulatory interactions utilized by human cells to control expression of amyloid-β precursor protein (APP) is still undefined.
Mre11 dimer flexibility, which may be regulated in cells to control MRN function, suggests new inhibitor design strategies for cancer intervention.
The objective of such procedures is to infuse a sufficient quantity of islet cells to control the level of blood glucose and, thereby, overcome the need for exogenous insulin.
Hall (1992) defined epigenetics as "the sum of the genetic and non-genetic factors acting upon cells to control selectively the gene expression that produces increasing phenotypic complexity during development" (1992, p. 89).
For instance, a self-assembled silica nanoparticle monolayer was employed to deliver negatively-charged RNA-based molecules (e.g. siRNA, miRNA) into neural stem cells to control neuronal differentiation (Fig. 3b) [61].
Post-transcriptional regulations of gene expression enable cells to control the contents of their proteomes.
In this context, the rapidity and efficacy of memory cells to control secondary infections are puzzling.
In vivo tumorigenic assay was performed to compare retroviral-mediated ERα knockdown cells to control shRNA-transduced cells.
However, in the HL-60 cells, menadione treatment significantly diminished oxygen consumption, perhaps due to the diminished ability of these cells to control cellular ROS levels (Figure 3B).
The ΔCPM was computed as the difference between the mean cpm of wells containing activated or resting A2b stimulator cells to control wells cultured with medium alone.
The same molecules interact in mammalian cells to control cell cycle progression, thus it appears that the same pathway can regulate mutually exclusive processes.
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