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BRD4 specifically governs the transcriptional elongation of stem cell transcripts by occupying their associated super-enhancers (SEs) and recruiting Mediator and CDK9 to those regulatory elements, thus emerging as a key regulator of the intricate gene expression network that maintains ESC identity.
These mRNAs could either be nurse cell transcripts imported into the meiotic oocyte nucleus or else the controversial instances of transcription from the meiotic nucleus (Saunders and Cohen, 1999; Cáceres and Nilson, 2005).
These mRNAs could either be nurse cell transcripts imported into the meiotic oocyte nucleus or else the controversial instances of transcription from the meiotic nucleus (Saunders and Cohen, 1999; Caceres and Nilson, 2005)." Reviewer #2: This manuscript presents a large scale in situ hybridization analysis of transcripts expressed during Drosophila oogenesis.
In summary, SAHA modulated numerous host cell transcripts, proteins and post-translational modifications of proteins, which would be expected to have very mixed effects on the induction of HIV-specific transcription and protein function.
Approximately 60% or more of all the alternatively spliced genes in T cells are differentially expressed at the mRNA transcript level during activation and only 25 30% of B cell transcripts.
This truncated form of the protein is generated from thymic transcripts that derive from the proximal promoter of the Lck gene, but not from peripheral T cell transcripts deriving from the distal promoter.
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ADMEM, however, promoted increased cell transcript and protein expression, as 5 of 9 growth factor combinations induced a 200% increase in collagen type I versus equivalent DMEM cultures.
It was formerly known as natural killer cell transcript 4 [3].
In summary, we conclude that zygotic division results in a divergence in apical and basal cell transcript profiles.
Interleukin-32 (IL-32), previously called natural killer cell transcript 4, has been recognized as a pro-inflammatory cytokine recently.
The results confirmed the suitability of the assay for single cell transcript quantitation and show its applicability to both abundant and rare transcripts (Figure S2).
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