Sentence examples for cell prevalence from inspiring English sources

Sections stained with PAS were evaluated for goblet cell prevalence, and Masson's trichrome-stained sections were used to detect Paneth cells.

We confirmed this latent PCV2-infected cell prevalence in a larger study of pig fetuses (data not published).

In contrast to in vivo effects, rapamycin significantly reduced CD11b+CD11c+ dendritic cell prevalence although they acquired activated phenotypes (Fig. S10A) similar to in vivo and genomic data.

We show that leukocyte recruitment to the laser-induced injury is independent of local DLL4 signaling, as IBA-1 positive macrophage/microglial cell prevalence in PBS and sDLL4/28-525 at 4 days were comparable.

More broadly, the dependence of stem cell kinetics on cluster formation, made possible by focal separation, may inform our understanding of how stem cell prevalence and distribution relate to tumour growth overall.

Thus, as a reflection of stromal cell prevalence, the stromal genes are more highly expressed in non-neoplastic tissue than in the paired tumor tissue; all mean fold difference values were 1.7-fold or greater in non-neoplastic tissues.

Three years following the accident, exposed subjects were compared to residents of a nearby unexposed community for the following immunological parameters: white blood cell count, total lymphocyte count, percent and total lymphocyte subsets (CD5, CD4, CD8, CD19, CD25, and CD26 cells), prevalence of autoantibodies, and antibodies to HCHO-human serum albumin (HCHO-HSA) conjugate.

This study aimed to investigate in normal oral epithelium, hyperplasia, dysplasia, and invasive oral squamous cell carcinoma the prevalence of p53 protein immunoreactivity and of angiogenesis, and to determine the correlation between them.

Lost to follow-up patients were younger (median age [IQR], 34.86 [31.08 38.21] versus 37.26 [33.95 42.49] years; P<0.000), and were more likely to be intravenous drug users (62.3% versus 47.7%; P = 0.029), but did not differ with respect to baseline pVL, baseline CD4 cell counts, prevalence of AIDS at baseline, or immunological responses at 6 and 12 months of HAART initiation.

Should genomic aberrations have an effect on the cancer cell, their prevalence differences have to manifest themselves at the transcriptomic level of the pathway.

The GES is derived from the expression levels of 23 genes as well as age and sex; the genes are grouped into highly correlated meta-genes which reflect both biological processes and cell type prevalence [ 7, 9].