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Transmission in a cell may also be stopped entirely if no UE is present.
Furthermore, apoptosis pathway proteins in other organelles within the cell may also both modulate apoptosis and metabolism.
The exposed surface area of epithelium and/or the density of target cells in or just beneath the epithelium in the male versus the female genital tract may be sufficient to explain this difference, but the ability to spread after infection of the initial cell may also be selected (see the figure).
Depending on the nature of the epitope and type of cell, the aggregation of epitopes on the target cell may also trigger cellular responses [5], [6].
Further localization of MMP activity, e.g. to specific microdomains on the tip cell, may also increase local MMP concentrations and VEGF cleavage.
Our results provide evidence that trafficking of other proteins, organelles, or vesicles in the cell may also be disturbed, although this cannot be true for all proteins as LRP localization was unchanged in the H4-APP cells.
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The walls of cork cells may also contain lignin.
In the process, their cells may also be clearing away more defective molecules, so that the animals age more slowly.
In some instances, neighbouring cells may also engulf the degraded components.
Somatic cells may also be used to generate transgenic clones.
Other lung cells may also produce inflammatory mediators, particularly the epithelial cells.
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