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In some cases, GLP proteins co-localized with golgi marker proteins at cell margins (merged images).
Both isoforms were significantly enriched in the cell margins, but exhibited distinct localizations (Fig. 3A and 3B).
These cells exhibit morphology that includes regular cell margins and spacing patterns when examined by the light microscope.
Adherens junctions co-localize with apical determinants at D/V cell margins (Fig. 1C) [7], [8], [10] [10].
As the process begins, polarity determinants are asymmetrically distributed within the rectangular cells; the baso-lateral components are enriched on the anterior/posterior (A/P) cell margins directly opposite to apical polarity determinants, which are enriched on the dorsal/ventral (D/V) cell margins (Fig. 1 B and C).
Specific staining revealed a time-dependant accumulation of filamentous actin at the cell margins, and this is the first report providing evidence for actin rearrangement associated with B. pilosicoli colonization.
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If femtocells are located at the cell margin of the primary systems, there will be no degradation in terms of the capacity of femtocell users.
During mitosis, animal cells undergo a complex sequence of morphological changes, from retraction of the cell margin and cell rounding at the onset of mitosis to axial elongation and cytokinesis at mitotic exit.
Small particles are drawn into the organism's proximity by cilia-induced fluid currents at all times, whereas larger particles may be held immobile at a distance from the cell margin when the cell generates characteristic feeding currents in the surrounding media.
Collectively, these results support the hypothesis that 1) myosin IIA predominantly confers contractile and retrograde force at the cell margin, 2) myosin IIB predominantly confers protrusive force at the cell margin and 3) both myosin IIA and IIB contribute to overall lamellipodia extension on extracellular matrix.
The implication of this "advection hypothesis" is that transport and supply of actin monomer in these channels is an important regulatory influence on the motion of the cell margin.
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