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ACS-induced myocardial cell injury may also cause mitochondrial dysfunction.
Renal cell injury was investigated using 99mTc-lactadherin.
Glutamine (Gln) has been reported to repair myocardial cell injury.
Exposure to H2O2 induces apoptosis-like cell injury.
Posttreatment with isoflurane, but not desflurane, reduced this cell injury.
Cell injury releases actin, the most abundant cell protein.
Both ferroporphyrins completely protected from EFV-induced cell injury, suggesting that peroxynitrite contributed to the cell injury.
Although it is assumed that tumour cell injury is caused by osmotic shock, the exact mechanism of potential cell injury by distilled water is not known.
C-reactive protein can bind histones and reduce histone-induced endothelial cell injury.
Therefore, supernatant LDH of cultured HAECs is detected as a marker for cell injury [36].
It is now known that there are at least two basic patterns of cell injury progressing to cell death: cell injury with swelling, known as oncosis, and cell injury with shrinkage, known as apoptosis.
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