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The electrochemical experiments took place in a glass electrochemical cell; discs of Ti and bare and nitrided Ti-23Nb-0.7Ta-2Zr-0.5N asloy were used as the working electrodes connected with saturated calomel electrode (SCE) by a Luggin-Haber capillary; auxiliary electrode was a platinum plate.
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For the density of mitotic cells, discs were imaged on either a Leica TCS SL or a Zeiss LSM 700.
In type-II cells, DISC-induced caspase-3 activation is incomplete and insufficient to induce apoptosis.
In order to test PSR-3-283A PSR-3-283A PSR-3-283Atically help antibiotics kill bacterial cells, disc diffusion assays were conducted and zone diameters were measured for E. coli DH10B without or with pBC SK blaSHV-1 (Table 3).
Research suggests that a vertebral trauma and especially a fractured endplate can cause disc cell apoptosis and disc degeneration [ 19, 20].
Nerlich and colleagues [ 31] found no CD68-positive cells in discs of foetus, infants, or adolescents.
The source of cells for disc repair is not immediately apparent, as no benign site is known for harvesting authentic autologous disc cells, and it is preferable to avoid the use of allogeneic donor disc cells.
Degenerative disc disease (DDD), characterised by the deterioration and degradation of disc matrix, has been shown to affect disc cell mechanobiology, leading to the inability of disc cells to respond to physiological loads in the normal anabolic manner.
The hydrogen oxidation reaction (HOR) was determined using a half-cell rotating disc electrode in a 0.5 M H2SO4 electrolyte.
Ophthalmoscopic examination of both eyes showed vitreous cells, optic disc edema, small amounts of subretinal hemorrhage, and punctate choroidal lesions throughout the fundus.
We refer to the elongated flat cells as disc shaped and 7G).
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