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Complement is usually activated, leading to cell destruction.
Notably, infected mast cells promoted beta cell destruction.
Alternatively, the presence of these maternal antibodies may merely serve as a biomarker of cell destruction.
Caspases turn off cell-protective mechanisms and activate pathways that lead to cell destruction.
Excessive blood cell destruction caused by increased spleen function is important evidence of hypersplenism.
Anti-tumor cell antibodies should, in theory, activate complement resulting in cell destruction.
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But Dr. Ross also suggested that caffeine might somehow protect against the nerve-cell destruction that causes Parkinson's.
Rabinovitch, A. et al. Human pancreatic islet beta-cell destruction by cytokines is independent of nitric oxide production.
But by impeding hyaluronan synthesis, 4-MU re-establishes the induction of enough Tregs to prevent beta-cell destruction.
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