'cell demise' is a correct and usable term in written English.
It is a scientific term used to refer to the death of a cell. For example: The researchers studied the cell demise caused by various environmental factors.
Development of more potent and selective small-molecule drugs that activate ER Golgi mediated cell demise is also needed.
For decades, research has sought to unravel the molecular events mediating sensory cell demise, emphasizing the roles of reactive oxygen species and their potentials as therapeutic targets.
Above changes were related to altered cell demise (apoptosis and necroptosis), as shown by upregulated expression of cleaved caspase 9, cleaved caspase 3 and RIP1/RIP3/MLKL cascade.
Expanding our knowledge of the molecular "crosstalks" that regulate tumor cell demise is crucial in guiding the successful design of future anti-cancer therapeutics.
Tumor cell demise is an important event in the elimination of abnormal malignant cells and provides an important mechanism of natural tumor suppression.
Understanding the intricate crosstalk between autophagy and necrosis is essential, as this process, on the one hand, is required for necrosis while, on the other, can act as a pro-survival mechanism to inhibit cell demise.
To conclude, we show that both early Ca2+ influx and mitochondrial decay are conserved mechanisms of pyroptotic cell demise that are mediated by GSDMD and precede plasma membrane rupture following caspase-11 activation.
Taken together, our data provide evidence that metergoline elicited cell death process in C. krusei through elevation of the intracellular ROS level and perturbation of mitochondrial homeostasis, followed by damage of nucleus and eventual cell demise.
This releases into the cytosol key apoptogenic proteins that triggers cell demise via apoptosis [27, 28].
Unlike apoptosis, necrosis was recognized as the passive and unwanted cell demise committed in a non-regulated and disorganized manner.
The role of TG2 in cell demise is extremely controversial.
