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Stapyhylococcus aureus (S. aureus) is a major human pathogenic gram-positive bacterium causing a diverse array of diseases ranging from minor skin and wound diseases to more serious and life-threatening diseases like pneumonia, endocarditis and arthritis [ 1]. S. aureus, as all gram-positive bacteria, has a thick cell wall of peptidoglycan which covers a thin cytoplasmic membrane.
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Multidrug resistance is now common among the pathogenic Staphylococci and perhaps, greatest concern because of their intrinsic virulence, ability to cause a diverse array of life threatening infections and capacity to adapt to different environmental conditions [6].
Group A Streptococcus (GAS) causes a diverse array of human infections and sequelae, and exhibits a complex pathogenic behavior.
Pathogenic leptospires infection caused a diverse array of clinical manifestations ranging from subclinical infection to undifferentiated febrile illness to jaundice, renal failure, and potentially lethal pulmonary hemorrhage [2], [3].
Staphylococcus aureus (S. aureus) causes a diverse array of clinical conditions in humans, ranging from asymptomatic colonization to endocarditis and death.
Pleural effusions can be caused by a diverse array of pleural, pulmonary or extrapulmonary diseases [ 1, 2].
Motor neuron diseases (MNDs) are a heterogeneous group of neurodegenerative disorders that are caused by a diverse array of factors, including both genetic and sporadic.
Autism spectrum disorders (ASD) are paradoxical: the syndromes within this group present with a highly recognizable set of symptoms, yet they can be caused by a diverse array of genetic abnormalities and environmental insults, such as prenatal infection, hormonal exposure and teratogens (Newschaffer et al., 2007; Abrahams and Geschwind, 2008).
Boundary elements have a diverse array of functions.
The centriole/basal body protein SAS-6 normally regulates assembly and duplication of these organelles and its depletion causes severe flagellar/ciliary abnormalities in a diverse array of eukaryotes.
This finding indicates that, at least at the level of resolution offered by restriction fragment length polymorphism analysis, these antigens showed remarkable genetic homogeneity throughout the region sampled, perhaps caused by balancing selection to maintain a diverse array of antigen haplotyes.
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