In the current article we described this problem, presented an overview of possible causes, and we proposed solutions to increasing access to these services.
Our data support the hypothesis that HBoV1 may cause RTIs, and we propose that HBoV1 mRNA could be used with benefit, instead of HBoV1 DNA, as a diagnostic target.
Since the graph corresponding to a signaling network does not reflect the possible conditional dependency between incoming edges and cannot resolve inconsistent paths caused by inhibition, we propose an augmented graph representation that naturally incorporates synergy and inhibition.
As they are essentially equivalent phenomena, and thus have the same cause and features, we propose differentiating between them by considering that they refer to the two different levels of selection that appears in quasispecies models.
We postulated that the hypothalamic defect that we propose causes abnormalities of circadian rhythm and metabolism in Magel2-null mice could be accompanied by reduced fertility.
To prevent the deterioration caused by the process, we propose a selective averaging approach.
To corroborate the claim that our phase shifts are essentially caused by noise directivity, we propose a synthetic test with a homogeneous medium (constant phase speed of 2.45 km/s at 2-s period).
On the other hand, for zeolite supported metal catalysts that suffer from deactivation caused by pore blocking, we propose a strategy to avoid the deactivation problem by depositing metal nanoparticles exclusively on the external surfaces of support.
Figure 3 illustrates the recombination events that we propose caused these new recombinant tRNAs-Ser in B. pseudomallei K96243 and B. thailandensis E264.
Although we do see suppression of MMP-2 and MMP-9 expression, which is likely due to transcriptional downregulation caused by S3DN expression, we propose that Stat3 may also be acting at the membrane to enhance motility and invasion.
