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As shown in Fig. 2, RWE (150 µg/mL) caused increase in cellular ROS levels as evident by increased fluorescence by ROS sensitive dihydroethidium (DHE).
In mice, MeHg significantly caused increase in plasma insulin as well as C-peptides.
ZNS caused increase in the quantity of EAAC-1 protein in hippocampus and cortex and down regulation of the GABA transporter GAT-1.
The viscosity decreased with temperature arranged from 10 to 60 °C which caused increase in protein from 7.18 to 8.49 g/kg.
In addition, DLM caused increase in the malondialdehyde (MDA) level accompanied by decrease in antioxidant enzymes activity such as superoxide dismutase (SOD), catalase (CAT), glutathione peroxidase (GPx) and glutathione reductase (GR).
DIZ caused increase in the quantity of VGAT mRNAs and proteins, and down regulation of GABA transporters GAT-1 and GAT-3 mRNAs and proteins after the PTX re-kindling.
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The infection also caused increases in hepatic OGG1 transcript levels and NRF-1 and NRF-2α transcript and protein levels.
The researchers also found that increases in Rev-erbα caused increases in Cx43 concentrations when normal mice were awake.
Handgrip caused increases in plasma ADM, ET-1 and PRA only in older men (p < 0.05).
Strikingly, PKD2 loss caused increases in thymic output, lymphadenopathy and splenomegaly in TCR transgenic mice.
Changes in lifestyle among evacuees caused increases in body weight and blood pressure [ 10].
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