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In the Type II model, a 10-fold reduction of the dissociation probability caused a length compaction of over 40%, more than what is observed in vivo.
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This causes a length mismatch between and.
As shown in Figure 2d, when Vstage is larger than half of Vtip, the two regions machined in the adjacent AFM scanning cycles are nonoverlapping, which can cause a length of the unmachined region (LU) expressed by Equation 7.
The second mechanism that can cause a length disparity between an inserted segment and the fragment excised during splicing is when an inserted segment has an internal splice site and its unspliced terminus extends the length of its adjacent exon.
Strikingly, combined downregulation of Kif5B and Dync1h1 caused a greater length increase than with each siRNA alone, as predicted by the frequency-dependent model in the case of partial downregulation of both motors.
Application of a constant, comparatively high external load during continuous cooling caused a step-wise length decrease in the temperature range of the transformation instead of the step-wise length increase observed for the martensitic transformation in absence of an external load.
In addition, a shorter swing time caused a shorter step length in the uninvolved limb since there was less time to step forward.
In humans, knee joint contractures required more quadriceps force for stability, caused a shorter stride length, increased oxygen consumption and negatively impacted balance and risk of falling [ 26– 28].
Therefore, more digits in a column caused a larger RT for length 3 condition than for length 2 condition, resulting in the length effect.
Of particular interest, Gan et al. (2011) noted that length mutations affecting intron splice sites may activate latent splice sites in adjacent exons, thereby causing a compensatory length mutation in affected introns.
This in turn caused a reduction in sarcomere length, which we scaled up to give the expected cell length L as shown in Figure 5 d).
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