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As the PC is also involved in motor tasks, Experiment 1b (number naming) was conducted to exclude motor dysfunction as cause for deviations potentially observed in 1a.
The default prediction is that the level of genetic diversity on Z or X chromosome should be 75%% of that on autosomes, but it is very difficult to pinpoint the exact cause for deviations in empirical studies because of confounding genetic processes e.g., [ 30, 31].
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It can also provide audio 'turn-by-turn' directions, as well as even tactile feedback to provide proper instructions in case something happens during extra-vehicular activity (EVA) and there's cause for deviation from the standard plan.
The causes for deviations are however hard to identify by visual inspection and complicate the correction of a problem in the next cycle or batch.
Deviations from the calculated values are shown in the areas A and C. The main cause for these deviations is the assumption of frictionless contact in the model.
The immediate cause for this deviation from the trend is not known, and therefore warrants further investigation.
The likely cause for this deviation is that many of the sequence fragments were observed thousands of times more than expected (up to 95,014 reads from a single end of an ApeKI fragment).
Other possible causes for such deviations are unlikely, as small sample sizes appear unimportant when comparing with previously analysed larger samples [ 52], no recurrent amplification failure (null alleles) was observed in any locus or population, and no evidence supports the co-existence of distinct but sympatric sub-populations within estuarine patches (Wahlund effects).
The causes for guideline deviation are listed in Table 6.
The program Micro-checker [ 39] was used to investigate likely causes for possible deviation from Hardy-Weinberg equilibrium.
Possible causes for a deviation from the model of CSF absorption in some patients were a variation in formation rate or venous pressure or a pressure dependent Cout.
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