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The recycling of older C from dying roots to support seasonal growth of new roots could similarly cause C to remain in the biomass of fine roots for a considerably longer time than in individual roots themselves3,27.
On the other we have what might be called ennoblers: facts G such that the cause c would have not been required for e, had G nor obtained.
It does not cause (c) membrane asymmetry of cells as indicated by lack of Annexin V binding as well as (d) activation of caspase 3 and cleavage of PARP.
Apparently there are two kinds of factors "without which the cause would not be a cause". On the one hand, we have enablers: facts G such that the cause c would not have been enough for e, had G not obtained.
More specifically, the root cause c i associated to that RCP is the diagnosis for the given cell.
There are good reasons why, unlike the intrinsic properties of a cause c, its extrinsic properties cannot be causally efficacious in the process whereby c produces its effect.
Similar(31)
The c-myc gene is a known target of the Notch signaling pathway [7], and with Hes1 being downstream from Notch receptor signaling, it is therefore plausible that Hes1 can cause c-myc up-regulation.
The majority of Seipin mutations cause C-terminal truncations, along with a handful of point mutations.
The second goal arose because mammillothalamic tract lesions cause c-Fos hypoactivity in numerous sites including the hippocampus, retrosplenial cortex, and prelimbic cortex (Vann and Albasser, 2009).
Furthermore, a short 1 h exposure of NALM-6 cells to 1 μℳ JQ1, insufficient to cause c-Myc downregulation, also led to a marked reduction in fork progression rates.
Repeat-induced point mutation (RIP) is a gene silencing mechanism that can cause C-G to T-A mutations on repetitive DNA sequences, and the mutations from C to T mostly occur at CpA dinucleotides [ 31].
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