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Catheter colonization and bloodstream infection during the first week after insertion of a central venous catheter have been shown to result from the patient's own skin flora.
Catheter colonization.
In short-term catheterization, the exoluminal route is considered the main mechanism of catheter colonization.
Catheter colonization and catheter-related bloodstream infection (CRBSI) incidences were compared.
In addition, two recent studies showed an increased risk of Candida spp. catheter colonization [84, 85].
PM is responsible for the diagnosis area of catheter colonization and C-RBSI.
Catheter colonization risk was significantly higher in the 10 % PVI group.
Experimental work has demonstrated that the formation of a fibrin sheath surrounding the catheter greatly increases catheter colonization [71].
In rats, subcutaneous administration of the low-molecular-weight heparin enoxaparin decreases fibrin sheath formation and the incidence of catheter colonization with biofilm-producing S. epidermidis [72].
However, few studies investigated the superiority of 1 % CHG over either 0.5 % CHG or 10%% PVI for the prevention of catheter colonization as CDC guideline recommends.
Do not administer systemic antimicrobial prophylaxis routinely before insertion or during use of an intravascular catheter to prevent catheter colonization or CRBSI.
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