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In recent years, a number of cases have been attributed to EEHV1 among wild Asian elephants.
Approximately 50% of FAD cases have been attributed to mutations in three genes, amyloid precursor protein (APP) [1], presenilin-1 (PSEN1) [2] or presenilin-2 (PSEN2) [3].
About 11% of CRC cases have been attributed to overweight and obesity in Europe.
Five percent to 10% of Leigh syndrome cases have been attributed to mutations in the MT-ATP6 gene.
Among pigs, many serotypes cause severe infections, but nearly all human cases have been attributed to serotype 2 (1, 3 ).
The remaining 20% of cases have been attributed to mutations in a fourth unidentified CCM gene (Riant et al, 2010).
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Those cases had been attributed to a little-known virus called Zika, a member of a family of RNA viruses that includes dengue, West Nile, and yellow fever.
The necrosis found in such cases has been attributed to a polyketide (called mycolactone) that is produced by M. ulcerans and acts as an extracellular toxin (13 – 16 ).
The increase in the number of cases has been attributed to factors such as genetics, environmental pollution, urbanization and changing food habits.
1– 5 This increased number of cases has been attributed to various factors, including inefficacy of the Aedes aegypti eradication program, population growth, increased urbanization, and climatic changes.
The winter increase in cases has been attributed to cold weather, lower humidity, the crowding together of susceptible hosts, associated viral infections, and air pollution (1 – 3 ).
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CEO of Professional Science Editing for Scientists @ prosciediting.com