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Accompanied by changes in cardiac form and function, they manifest themselves in alterations of cardiac strains, fiber stretches, and muscle volume.
Specificity of the assay for the cardiac form of troponin-I was confirmed using samples spiked with canine skeletal muscle troponin-I.
We explore a generic bi-ventricular heart model in response to volume- and pressure-overload to demonstrate how local changes in cellular morphology translate into global alterations in cardiac form and function.
Restoration of cardiac form and function in post-infarct mIGF-1 transgenic mice was facilitated by modulation of the inflammatory response [30].
Cardiac form was the commonest form of presentation (60%).
From the 8 patients with the cardiac form, five presented the stage A, two the stage B and one the stage C of the cardiac form.
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The use of increasingly sensitive assays for the cardiac forms of Troponin I (cTnI) and Troponin T (cTnT) has revolutionized the care of patients presenting with suspected non-ST elevation acute coronary syndromes (NSTE-ACS) [ 70].
Decellularized myocardial matrix hydrogels derived from porcine cardiac tissue form a nanofibrous hydrogel once thermally induced at physiological temperatures.
Cardiac fibroblasts form one of the largest pools of cells in the heart and contribute to the normal structure and function of the myocardium (Souders et al., 2009).
In the healthy heart, cardiac myocytes form an electrical syncytium embedded in a supportive fibroblast-rich extracellular matrix designed to optimize the electromechanical coupling for maximal contractile efficiency of the heart.
To further evaluate the hypertrophic response, we performed histological analysis of the cardiac muscle form saline- and AngII-treated mice.
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