Sentence examples for capacity for maturation from inspiring English sources

Exact(3)

In conclusion, NECs at acute injury sites are a proliferative, transient cell population with capacity for maturation into astrocytes with possible neuronal differentiation observed in older injuries.

Experimental data indicate that the ameloblasts in the hypomineralized enamel are capable of forming an enamel of normal thickness, but with a substantial reduction of their capacity for maturation of enamel [ 11].

In this study, the authors examined the capacity for maturation of iPSDCs compared with that of BMDCs in addition to the capacity for migration of iPSDCs to regional lymph nodes.

Similar(57)

Preterm babies show specific glomerotubular imbalance because GFR sharply increases after birth while tubular secretion matures slower, but more steadily, to result in a proportional higher capacity for postnatal maturation of renal tubular function [ 42].

There is great capacity for tubular maturation with FeNa approximating term babies at 28 days of life.

Recent data suggest that, compared with maturational patterns in term neonates, the maturational creatinine clearance in preterm neonates matures slower, while there is already a higher capacity for postnatal tubular maturation in preterm neonates and at the age of 1 month [ 42].

Several unique features of neuroblastoma (NB), including the capacity for spontaneous regression and maturation to benign pathology, suggest that genes that regulate cellular proliferation, survival and differentiation may be involved in directing clinical tumour aggressiveness.

Mechanistic studies revealed that Jak2/SATA5 signaling is pivotal for DC development and maturation, while the capacity for DCs secretion of proinflammatory cytokines is regulated by both Jak2/STAT5 and Jak2/STAT6 signaling.

Hypothesized mechanisms for age-related differences include maturation of the immune system or links between host cell maturation and the capacity for virus replication [22].

In addition, it has been shown that in the absence of IGF-I [19] or PDGF-A [20], there is a failure of Leydig cell maturation, and reduced capacity for testosterone production.

However, Atoh1 induction in adult mice cannot facilitate the formation of hair cell-like cells, which suggests that cochlear-supporting cells lose their cellular pliancy and capacity for cellular reprogramming during inner ear maturation.

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