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There was significantly more HER-2/neu oncoprotein immunoreactivity in BPH than in areas of CaP (P < 0.001).
This ratio can be expressed as ψ = 1 - P cap P simul (17).
And if the number of defective products in the random sample is lower than (alpha_{1}), we will have: overline{E}_{3} (p) = Pr (theta le alpha_{1} cap p le p_{1} )E_{p le p1} left[ p right] + Pr (theta le alpha_{1} cap p_{1} le p le p_{2} )E_{{p_{1} le p le p2}} left[ p right] + Pr (theta le alpha_{1} cap p ge p_{2} )E_{{p ge p_{2} }} left[ p right] (9).
There was no statistically significant difference in size distribution between the 40 LGD polyps, 40 HGD polyps or the 40 CaP (LGD vs. HGD, p = 0.548; HGD vs. CaP, p = 0.397; LGD vs. CaP, p = 0.068).
Significance was borderline for professional groups in CAP (p = 0.025).
On the contrary, CoPEP did not distinguish complicated from uncomplicated CAP (p = 0.95).
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Acute respiratory distress syndrome occurred in 20 CAP women (28.2 vs. 3.7% for non-CAP; p < 0.0001), and 6 required extra-corporeal membrane oxygenation (8.4 vs. 4.5% for non-CAP; p = 0.23).
Preterm extraction occurred for maternal reasons in 15 CAP women (21.1 vs. 7.8% for non-CAP; p = 0.004), and more frequently by cesarean section (84.2 vs. 53% for non-CAP; p = 0.0003).
Fetal survival did not differ significantly, as live births occurred in 85 vs. 77.4% for non-CAP (p = 0.39).
The BV/TV at week 12 was significantly higher than that at week 4 in both groups (control: P = 0.042; nano-CaP: P = 0.005) (Table 2).
Reactivity against the native counterpart of CCP (anti-CAP) was, compared with HC, increased in RA patients for IgG anti-CAP (p < 0.01) and in CF patients for IgA anti-CAP (p < 0.05), although this was not necessarily reflected in increased seropositivity (Fig. 2).
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