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Tension-type headache is far more prevalent than migraine and for that reason possible genetic mechanisms cannot be elucidated with a genetic epidemiological survey [3, 4].
The exact nature of these changes cannot be elucidated with human in vivo studies.
While some chemicals may have beneficial effects, others may have detrimental effects in individuals with autoimmune predisposition, and the adverse consequences of this sum of exposures cannot be elucidated with the information available.
Overall, we believe that despite these limitations, our study shows that incorporation of fine-scale anatomical complexity, such as blood vessels and endocardial structures, can provide insight into novel electrophysiological mechanisms that cannot be elucidated with previously used simplified models.
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Why lifespan is shifted this way cannot be elucidated at this stage, but it seems consistent with some compensation of inbreeding depression.
The NETCAR algorithm is a powerful CAR mining algorithm that can be used to extract relevant genes (COGs) associated with a phenotype observation, that cannot be elucidated by simple pairwise comparisons.
Taken together, it is becoming clear that establishment of a predictive role of KRAS mutation for treatment of mCRC with chemotherapy and biological agents, including bevacizumab, cannot be elucidated without evaluation of specific KRAS mutations.
However, most of these studies were with cross-sectional study design, the time course of metabolic side effects and SMD cannot be elucidated without a cohort study.
However, the process of formation of non-extractable residues cannot be elucidated by this approach.
However, the exact role of these conserved elements cannot be elucidated without structural and physiochemical information.
However a clear lower boundary cannot be elucidated from this data set as we did not observe the increase in incidence of RBC transfusion we expected to see with extremely low Hb levels.
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